Peptic Ulcer

Overview

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A peptic ulcer is an open sore or lesion that forms on the inner lining of the stomach or the upper part of the small intestine (duodenum). It results from the erosion of the protective mucosal lining due to excessive stomach acid or infection. Peptic ulcers are categorized into two main types: gastric ulcers, which occur in the stomach, and duodenal ulcers, which are found in the duodenum.

The condition is fairly common and can affect individuals of all age groups, though it's more frequently diagnosed in adults. While earlier it was believed that stress and spicy foods were the primary causes, it is now well-established that the majority of peptic ulcers are caused by Helicobacter pylori (H. pylori) infection or prolonged use of nonsteroidal anti-inflammatory drugs (NSAIDs).

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Symptoms

The symptoms of peptic ulcer disease (PUD) vary depending on the location and severity of the ulcer. Some individuals may remain asymptomatic until complications arise. Common symptoms include:

  • Burning stomach pain: This is the hallmark symptom, typically felt between the breastbone and the navel. It often occurs when the stomach is empty and may improve temporarily after eating or taking antacids.
  • Bloating and belching
  • Nausea or vomiting
  • Loss of appetite
  • Weight loss
  • Heartburn
  • Dark or tarry stools (due to gastrointestinal bleeding)
  • Vomiting blood or material that looks like coffee grounds
Complications

If left untreated, peptic ulcers can lead to serious complications:

  • Bleeding: The ulcer can erode blood vessels, leading to internal bleeding. This can cause anemia or life-threatening hemorrhage.
  • Perforation: An ulcer can burn through the stomach or intestinal wall, causing a perforation, which is a medical emergency.
  • Gastric outlet obstruction: Swelling or scarring from ulcers can block the passage of food, leading to vomiting and weight loss.
  • Peritonitis: Inflammation of the peritoneum due to perforation, which is often fatal if not treated immediately.
  • Increased risk of gastric cancer: Chronic H. pylori infection may increase the risk of developing stomach cancer over time.
Causes

The most common causes of peptic ulcers include:

  • H. pylori infection: This bacterium damages the mucous coating of the stomach and duodenum, allowing acid to irritate the lining.
  • NSAIDs: Prolonged use of ibuprofen, aspirin, or naproxen can inhibit the stomach’s ability to protect itself from acid.
  • Excess stomach acid: Conditions like Zollinger-Ellison syndrome result in increased acid production that can lead to ulcers.
  • Smoking and alcohol consumption: These can impair mucosal defense and promote ulcer formation.
  • Stress and diet: Though not direct causes, they can exacerbate symptoms and delay healing
Prevention

Preventive measures can significantly reduce the risk of developing peptic ulcers:

  • Avoid or limit NSAID use: If necessary, take them with food or use protective agents like proton pump inhibitors (PPIs).
  • Quit smoking: Tobacco use increases stomach acid and reduces mucosal protection.
  • Limit alcohol intake: Alcohol irritates and erodes the stomach lining.
  • Practice good hygiene: Since H. pylori spreads through contaminated food or water, washing hands and consuming safe food are essential.
  • Manage stress: Chronic stress may not cause ulcers but can worsen symptoms and hinder recovery.
Risk Factors

Several factors increase the likelihood of developing a peptic ulcer:

  • Infection with H. pylori
  • Long-term NSAID use
  • Age above 50 years
  • Smoking
  • Alcohol abuse
  • Family history of ulcers
  • Certain medical conditions (e.g., liver, kidney, or lung diseases)
  • Frequent use of corticosteroids, especially when combined with NSAIDs
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How is it Diagnosed?

Peptic ulcers are open sores that develop on the inner lining of the stomach (gastric ulcers) or the upper portion of the small intestine (duodenal ulcers). The diagnosis begins with a detailed clinical history, including symptoms such as burning epigastric pain, bloating, nausea, or early satiety. Pain that improves or worsens with eating can help differentiate gastric from duodenal ulcers.

A physical examination may reveal mild tenderness in the epigastric region. However, definitive diagnosis requires investigations. The urea breath test, stool antigen test, or blood test for Helicobacter pylori is commonly used since H. pylori infection is a major causative factor.

Upper gastrointestinal (GI) endoscopy is the gold standard for diagnosis. It allows direct visualization of the ulcer and biopsy if needed, especially to rule out malignancy in gastric ulcers. In some cases, a barium swallow X-ray may be used, although it is less commonly preferred due to lower sensitivity.

In patients taking NSAIDs, correlation with medication history is critical. Further investigations like complete blood count (CBC) may detect anemia if there’s chronic bleeding. In complicated cases (e.g., perforation), imaging like abdominal X-ray or CT scan helps identify free air or inflammation.

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FAQs

Some mild ulcers can heal temporarily, but without treating the underlying cause (e.g., H. pylori), they are likely to recur.

H. pylori infection can be transmitted through contaminated food, water, or saliva, but the disease itself is not contagious.

Stress alone does not cause ulcers but can worsen symptoms or delay healing in individuals already affected.

There’s no specific "ulcer diet," but patients should avoid foods and drinks that irritate the stomach, such as spicy foods, caffeine, and alcohol.

Tests include urea breath test, stool antigen test, blood antibody test, and gastric biopsy during endoscopy.

Surgery is now rare and is only used in cases of complications like bleeding, perforation, or obstruction.

With proper treatment, ulcers usually begin to heal within a few days and can resolve completely in 4 to 8 weeks.

Yes, especially if H. pylori is not completely eradicated or if NSAID use continues.

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