Achalasia

Overview

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Achalasia is a condition that affects the esophagus, specifically the muscle at the lower end of the esophagus (LES), which fails to open properly to allow food and liquids to pass into the stomach. This results in a buildup of food and fluid in the esophagus, causing discomfort and other symptoms. Achalasia typically progresses slowly and can lead to significant discomfort and complications if untreated.

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Symptoms
  • Difficulty Swallowing (Dysphagia): Trouble swallowing both solids and liquids.
  • Regurgitation: The sensation of food or liquid coming back up from the esophagus, often without nausea.
  • Chest Pain: Discomfort or pain in the chest, which can sometimes be mistaken for heart pain.
  • Weight Loss: Due to difficulty eating and swallowing.
  • Coughing or Aspiration: Coughing or choking on food, which can lead to aspiration pneumonia.
  • Halitosis: Foul-smelling breath due to food stagnation
Complications
  • Esophageal Cancer: Long-term irritation and inflammation can increase the risk of developing esophageal cancer.
  • Aspiration Pneumonia: Food or liquid that enters the lungs due to regurgitation.
  • Esophageal Perforation: Rare but serious complication where a tear develops in the esophageal wall.
  • Severe Weight Loss: Due to malnutrition and difficulty eating.
Causes

The exact cause of achalasia is not well understood, but it is thought to be related to damage or degeneration of the nerves in the esophagus. Theories include

  • Autoimmune Reaction: The immune system mistakenly attacks the nerve cells controlling the esophagus.
  • Genetic Factors: There may be a genetic predisposition, though this is not well established.
  • Viral Infections: Certain viral infections may be implicated in nerve damage, though this is speculative.
Prevention
  • Early Diagnosis: While there is no known way to prevent achalasia, early diagnosis and management can help prevent complications.
  • Regular Check-Ups: For individuals with symptoms or a family history, regular monitoring and early intervention can help manage symptoms effectively.
Risk Factors
  • Age: Achalasia can occur at any age but is often diagnosed in people between the ages of 25 and 60.
  • Family History: There may be a slightly increased risk if there is a family history of the condition.
  • Autoimmune Conditions: People with autoimmune disorders might have a higher risk.
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How is it treated?

Treatment aims to relieve symptoms and improve esophageal function. Options include

Medications

  • Nitrates: Help relax the LES muscle to ease swallowing.
  • Calcium Channel Blockers: Can also help relax the LES.
  • Botox Injections: Botox can be injected into the LES to temporarily paralyze the muscle and improve relaxation.

Procedures

  • Pneumatic Dilation: A balloon is inserted and inflated to widen the LES, improving its ability to open.
  • Endoscopic Injection: Botox or other agents can be injected into the LES to relax the muscle.

Surgery

  • Heller Myotomy: Surgical procedure where the muscle of the LES is cut to allow easier passage of food and liquids. This can be performed laparoscopically.
  • Peroral Endoscopic Myotomy (POEM): A minimally invasive endoscopic technique to cut the LES muscle.

Lifestyle and Dietary Adjustments

  • Eating Small, Frequent Meals: To make swallowing easier.
  • Chewing Food Thoroughly: To aid in easier passage through the esophagus.
  • Eating Soft Foods: To reduce difficulty in swallowing.
  • Managing achalasia effectively usually requires a multidisciplinary approach, including gastroenterologists, surgeons, and sometimes dietitians, to address symptoms and improve quality of life. If you suspect you have achalasia or have symptoms, it’s important to consult with a healthcare professional for proper diagnosis and treatment.
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How is it Diagnosed?

Achalasia is a rare esophageal motility disorder characterized by impaired relaxation of the lower esophageal sphincter (LES) and absence of peristalsis in the esophageal body. Diagnosis typically follows a history of progressive dysphagia (for solids and liquids), regurgitation, chest pain, and weight loss.

Initial evaluation often involves a barium swallow study, which reveals a classic “birdbeak” appearance at the LES with esophageal dilatation. This test is non-invasive and helps identify anatomical abnormalities.

The gold standard for diagnosis is esophageal manometry, which measures esophageal pressure. Findings include incomplete LES relaxation and absence of normal peristalsis. High-resolution manometry can classify achalasia into three subtypes, which can influence treatment decisions.

Upper GI endoscopy is done to rule out malignancy at the gastroesophageal junction (pseudoachalasia) and to exclude mechanical obstructions or inflammation. While the esophagus may appear dilated with retained food or saliva, the LES may not relax appropriately during the procedure.

In certain cases, chest X-ray or CT scan may show a widened mediastinum or air-fluid levels if the esophagus is significantly dilated. These are especially helpful in advanced disease.

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